new discovery Shaking the medical world worldwide After the research reported it Memory loss of “Alzheimer’s disease”. It can be transmitted from person to person. If the hormones are received from a deceased donor, the pituitary gland
Summary of explanations from neurology specialist University College London Head of the Center for Prion Diseases “Prof. John Collinge” on Alzheimer’s disease infection in patients Having said that Alzheimer’s infection (Alzheimer’s) does not have the same characteristics of a viral or bacterial epidemic as the common cold or COVID-19. Alzheimer’s disease is a person who has had brain surgery using cells or tissues that have precursors to increase the chance of causingamnesia It is also a very rare event.
Furthermore, data from this research showed us that prion disease is similar to Alzheimer’s disease, which is characterized by the spread of proteins throughout our brain system
Prion disease is caused by an abnormality that spreads in the brain. caused by a protein infection And often occurs naturally, without any stimulation from external factors.
Data reveals that at least 1,848 people with Alzheimer’s disease in the UK received growth hormone between 1959 and 1985. It is extracted from the pituitary gland of a corpse or from the body of a deceased person.
Even in 1985, after it emerged that some patients had died from a type of prion disease called Creutzfeldt-Jakob encephalopathy, Creutzfeldt-Jakob disease (CJD) resulted from contamination of the hormone used with the protein that causes CJD, for which treatment was eventually stopped.
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There have been 80 reported cases in which beta-amyloid protein was found. (Amyloid-Beta) in the brain after death. But it is not yet clear whether there is a precursor to the development of Alzheimer’s disease. But other research indicates that all of us could be at risk of developing Alzheimer’s disease if we are stimulated by it. Amyloid beta is present in some hormones.
The researchers reported findings from eight people referred to the National Prion Clinic between 2017 and 2022.
All received cadaveric human growth hormone, but none had Alzheimer’s disease, while five patients had dementia that met clinical criteria for Alzheimer’s disease. With onset at age 38, three patients had brain scans consistent with the diagnosis, while two had biomarkers that met criteria for Alzheimer’s disease.
Of the other three patients, one had mild cognitive impairment. One person reported having intellectual difficulties. and have no such symptoms The first showed autopsy findings consistent with Alzheimer’s disease. and the latter met biomarker criteria for the disease.
Five patients had DNA data available, but only one showed genetic risk factors for late-onset Alzheimer’s disease. And no one knows that there is a genetic variation that causes early-onset Alzheimer’s disease.
The researchers added that Patients show some symptoms that differ from typical symptoms of Alzheimer’s disease. This occurs naturally or is linked to genetic risk. It is assumed that this symptom may be caused by diseases of different origins or by different “strains” of Beta-amyloid
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Reference: www.theguardian.com
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